EAE models

Abnormal NMDA receptor function exacerbates experimental autoimmune encephalomyelitis

Neuroscience / Multiple sclerosis / Inflammation / Neurodegeneration / Glutamate / Mice / Female / Animals / Neuroinflammation / EAE models / SYNAPSES / Voltage-Gated Sodium Channels / NMDA Receptors / Experimental Autoimmune Encephalomyelitis / Excitotoxicity / Glutamic Acid / Excitatory Postsynaptic Potentials / Mice / Female / Animals / Neuroinflammation / EAE models / SYNAPSES / Voltage-Gated Sodium Channels / NMDA Receptors / Experimental Autoimmune Encephalomyelitis / Excitotoxicity / Glutamic Acid / Excitatory Postsynaptic Potentials

Abnormal NMDA receptor function exacerbates experimental autoimmune encephalomyelitis

Neuroscience / Multiple sclerosis / Inflammation / Neurodegeneration / Glutamate / Mice / Female / Animals / Neuroinflammation / EAE models / SYNAPSES / Voltage-Gated Sodium Channels / NMDA Receptors / Experimental Autoimmune Encephalomyelitis / Excitotoxicity / Glutamic Acid / Excitatory Postsynaptic Potentials / Mice / Female / Animals / Neuroinflammation / EAE models / SYNAPSES / Voltage-Gated Sodium Channels / NMDA Receptors / Experimental Autoimmune Encephalomyelitis / Excitotoxicity / Glutamic Acid / Excitatory Postsynaptic Potentials
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